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Mitochondrial Metabolism

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Mitochondrial Metabolism

Chemical Structure Cat. No. Product Name CAS No.
Mitochondrial fusion promoter M1 Chemical Structure
BCP48945 Mitochondrial fusion promoter M1 219315-22-7
Mitochondrial fusion promoter M1 is a mitochondrial dynamic modulator that increases the expression of mitofusin 2, a mitochondrial outer membrane fusion protein, reduces brain mitochondrial dysfunction.
DX3-213B Chemical Structure
BCP46463 DX3-213B 2749555-66-4
DX3-213B is a highly potent, orally active oxidative phosphorylation (OXPHOS) complex I inhibitor. DX3-213B impairs ATP generation, and blocks MIA PaCa-2 cell growth.
Mito-apocynin (C11) Chemical Structure
BCP46038 Mito-apocynin (C11) 1254044-38-6
Mito-apocynin (C11), an orally active mitochondria-targeted triphenylphosphonium (TPP)-based compound, is synthesized by conjugating the Apocynin moiety with a TPP+ cation.
L-Histidine Chemical Structure
BCP45568 L-Histidine 71-00-1
L-Histidine is an essential amino acid for infants. L-Histidine is an inhibitor of mitochondrial glutamine transport.
NL-1 Chemical Structure
BCP45211 NL-1 188532-26-5
NL-1 is a mitoNEET inhibitor with antileukemic effect.
MitoTEMPO Chemical Structure
BCP43473 MitoTEMPO 1334850-99-5
Mito-TEMPO is a mitochondria-targeted superoxide dismutase mimetic with superoxide and alkyl radical scavenging properties.
Mito-LND Chemical Structure
BCP42814 Mito-LND 2361564-49-8
Mito-LND is an orally active and mitochondria-targeted inhibitor of oxidative phosphorylation (OXPHOS).
CCI-006 Chemical Structure
BCP40217 CCI-006 292053-42-0
CCI-006 is a selective inhibitor and chemosensitizer of MLL-rearranged leukemia cells, by inhibits mitochondrial respiration resulting in insurmountable mitochondrial depolarization and a pro-apoptotic unfolded protein response (UPR) in a subset of MLL-r leukemia cells.
CTPI-2 Chemical Structure
BCP37418 CTPI-2 68003-38-3
CTPI-2 is a specific SLC25A1 inhibitor.
Mitochonic Acid 5 Chemical Structure
BCP34060 Mitochonic Acid 5 1354707-41-7
Mitochonic acid 5 binds mitochondria and ameliorates renal tubular and cardiac myocyte damage. Mitochonic acid 5 modulates mitochondrial ATP synthesis.
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